Longevity & Aging Series (S1, E2): Dr. Steve Horvath's Special Collection in Aging | Aging-US

In the second installment of the Longevity & Aging Series, Dr. Steve Horvath, Professor of Human Genetics and Biostatistics at UCLA, and Principal Investigator at Altos Labs, revisits papers he co-authored that were published by Aging (Aging-US) and featured as a special collection of his research. #aging #interview Special Collection - https://www.aging-us.com/special-coll... Author contact - Steve Horvath - [email protected] Papers - DNA-methylation-based telomere length estimator: comparisons with measurements from flow FISH and qPCR https://doi.org/10.18632/aging.203126 Epigenetic mutation load is weakly correlated with epigenetic age acceleration https://doi.org/10.18632/aging.103950 Blood DNA methylation sites predict death risk in a longitudinal study of 12, 300 individuals https://doi.org/10.18632/aging.103408 Epigenome-wide association study of leukocyte telomere length https://doi.org/10.18632/aging.102230 DNA methylation-based estimator of telomere length https://doi.org/10.18632/aging.102173 Optimism is not associated with two indicators of DNA methylation aging https://doi.org/10.18632/aging.102090 Placental epigenetic clocks: estimating gestational age using placental DNA methylation levels https://doi.org/10.18632/aging.102049 Rapamycin retards epigenetic ageing of keratinocytes independently of its effects on replicative senescence, proliferation and differentiation https://doi.org/10.18632/aging.101976 Epigenetic clock analysis of human fibroblasts in vitro: effects of hypoxia, donor age, and expression of hTERT and SV40 largeT https://doi.org/10.18632/aging.101955 DNA methylation GrimAge strongly predicts lifespan and healthspan https://doi.org/10.18632/aging.101684 Cell and tissue type independent age-associated DNA methylation changes are not rare but common https://doi.org/10.18632/aging.101666 A multi-tissue full lifespan epigenetic clock for mice https://doi.org/10.18632/aging.101590 Epigenetic ageing is distinct from senescence-mediated ageing and is not prevented by telomerase expression https://doi.org/10.18632/aging.101588 Epigenetic clock for skin and blood cells applied to Hutchinson Gilford Progeria Syndrome and ex vivo studies https://doi.org/10.18632/aging.101508 An epigenetic biomarker of aging for lifespan and healthspan https://doi.org/10.18632/aging.101414 Leukocyte telomere length, T cell composition and DNA methylation age https://doi.org/10.18632/aging.101293 Accelerated epigenetic aging in Werner syndrome https://doi.org/10.18632/aging.101217 An epigenetic aging clock for dogs and wolves https://doi.org/10.18632/aging.101211 Longitudinal study of surrogate aging measures during human immunodeficiency virus seroconversion https://doi.org/10.18632/aging.101184 Epigenetic clock analysis of diet, exercise, education, and lifestyle factors https://doi.org/10.18632/aging.101168 Specific premature epigenetic aging of cartilage in osteoarthritis https://doi.org/10.18632/aging.101053 DNA methylation-based measures of biological age: meta-analysis predicting time to death https://doi.org/10.18632/aging.101020 Huntington's disease accelerates epigenetic aging of human brain and disrupts DNA methylation levels https://doi.org/10.18632/aging.101005 Epigenetic age of the pre-frontal cortex is associated with neuritic plaques, amyloid load, and Alzheimer’s disease related cognitive functioning https://doi.org/10.18632/aging.100864 Decreased epigenetic age of PBMCs from Italian semi-supercentenarians and their offspring https://doi.org/10.18632/aging.100861 Increased epigenetic age and granulocyte counts in the blood of Parkinson's disease patients https://doi.org/10.18632/aging.100859 DNA methylation age of blood predicts future onset of lung cancer in the women's health initiative https://doi.org/10.18632/aging.100809 Epigenetic age analysis of children who seem to evade aging https://doi.org/10.18632/aging.100744 The cerebellum ages slowly according to the epigenetic clock https://doi.org/10.18632/aging.100742 About Aging-US Launched in 2009, Aging-US publishes papers of general interest and biological significance in all fields of aging research and age-related diseases, including cancer—and now, with a special focus on COVID-19 vulnerability as an age-dependent syndrome. Topics in Aging-US go beyond traditional gerontology, including, but not limited to, cellular and molecular biology, human age-related diseases, pathology in model organisms, signal transduction pathways (e.g., p53, sirtuins, and PI-3K/AKT/mTOR, among others), and approaches to modulating these signaling pathways. Please visit https://www.Aging-US.com​​ or connect with us: SoundCloud -   / aging-us   Facebook -   / agingus   Twitter -   / agingjrnl   Instagram -   / agingjrnl   YouTube -    / agingus​   LinkedIn -   / aging